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Myotoxic effects of clenbuterol in the rat heart and soles muscle, Jatin G. Burniston, Journal of Applied PhysiologyPublished 1 November 2002 Vol. 1824-1832 ABSTRACT Myocyte-specific necrosis in the heart and soleus muscle of adult male Wistar rats was investigated in response to a. The β 2 ‐adrenergic receptor (AR) agonist, clenbuterol, has been widely reported as an anabolic agent capable of inducing pronounced muscle hypertrophy. 6 , 11 , 28 , 36 , 37 However, data from our laboratory have revealed that this agent is also capable of inducing significant myocyte apoptosis 4 and necrosis 3 in the heart and slow‐twitch soleus muscle of rats. Banned Respiratory Treatment By Kristin Hayes, RN Updated on August 03, 2022 Medically reviewed by Farah Khan, MD Clenbuterol is a beta agonist. In some parts of the world, it is used to treat breathing difficulties caused by conditions such as asthma or chronic obstructive pulmonary disease (COPD). The incidence of cardiomyocyte death increased in line with the dose of clenbuterol administered and was maximal (0. 2 % apoptosis and necrosis, respectively; <0. 05) in response to 5 mg of clenbuterol kg Fig. 3A Dose dependent clenbuterol-induced apoptosis and necrosis. 017 Abstract Background: High-dose clenbuterol (a selective beta2-adrenergic agonist) has been proposed to promote myocardial recovery during left ventricular assist device (LVAD) support, but its effects on cardiac and skeletal muscle are largely unknown. PMCID: PMC1831534 DOI: 10. 20407 Abstract We have investigated the dose- and time-dependency of myocyte apoptosis and necrosis induced by the beta2-adrenergic receptor agonist, clenbuterol, with the aim of determining whether myocyte apoptosis and necrosis are two separate processes or a continuum of events This alone should be enough to raise red flags for individuals considering taking the drug, clenbuterol heart cell death.
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